|
|
|||
|
chat transcript for anatomy (neuro and histo)
21:09:56 [Step_1] which tract is for voluntary refined movements of the distal extremities?
21:10:59 Lorena enters this room 21:11:13 [Step_1] hi lorena.....great to see you 21:11:28 [Lorena] 21:11:32 [Lorena] hi everybody 21:11:44 [hutals] hi lorena 21:11:45 [butter] hi 21:12:17 [Lorena] just came to say hi and will join you for sure next one, i miss you guys!! 21:12:18 [Step_1] for those who dont know, lorena is one of the people who helped get these chats started 21:12:32 [Step_1] we miss you too lorena 21:13:14 [Lorena] i am so happy the chats continue so i dont distract you more , see you soon 21:13:38 [Lorena] study and enjoy your studies, best of luck again in your score step 1!! 21:13:42 [Step_1] thanks for stopping by. see ya in the next chat 21:13:50 [Step_1] thanks! 21:14:01 [Lorena] nice to see you again hutals, bye 21:14:06 [hutals] bye lorena 21:14:32 [hutals] i think the answer is corticospinal tract. is that right? 21:15:01 [Step_1] lorena is great. you guys will gain alot from her participation 21:15:27 [Step_1] yes hutals, the answer is corticospinal tract...good job 21:16:03 [Step_1] what is the difference between UMN and LMN signs? 21:17:34 [butter]>[Step_1] flaccid muscle in LMN, Barbinski and hyperreflex in UMN, 21:18:02 [Step_1] UMN has everything increased and LMN has them decreased.....so UMN has spastic paralysis, hyperreflexia, pos babinski, inc muscle tone, etc while LMN has flaccid paralysis, areflexia, no babinski, etc....good job butter 21:18:10 [ayouh] in umn everything is up, spastic paralysis, Hyperreflexia, +ve babinski, large area involved, and increased muscle tone....where everything is down is lmn 21:18:42 [ayouh] same way i memorized it step 1...cooll... 21:18:44 [Step_1] yes, very good ayouh 21:18:56 [butter] muscle fasciculation appear in UMN or LMN? 21:19:03 [freaha] hi every one 21:19:13 [butter] hi freaha 21:19:17 [Step_1] hi freaha 21:19:20 [freaha] in LMN 21:19:26 [hutals] hi freaha 21:20:26 [Step_1] LMN 21:20:42 [Step_1] sorry, didnt see the question until now 21:21:12 [freaha] well ...in a lesion of bulbospinal tract which cranial nerves show problems??????? 21:21:51 [butter] facial nerve VII 21:22:08 [freaha] and 21:22:40 [Step_1] 8? 21:22:49 [butter] i thought all others get bilateral control except contralateral lower face 21:23:04 [butter] yeh decreased sound localization 21:23:33 [freaha] facial and hypoglossal nerve 21:24:16 [Step_1] good one 21:24:27 [butter] why XII? 21:25:45 [Step_1] question described a pt who stubbles in the dark, but is fine in daylight. what do you think it might be? 21:26:16 [butter] vestibulo prob? 21:26:58 [Step_1] hint.....when eyes closed and fall over, this is pos rhombergs, so dorsal column. when eyes open and fall over, this is cerebellar 21:26:58 [freaha] nightblindness 21:27:27 [freaha] ohh 21:27:47 [butter] i c 21:28:51 [hutals] thats a good point 21:29:29 [butter] where is the cell bodies of corticospinal tract? 21:30:01 [Step_1] sorry, my computer is slow.....since it is dark, it is the same as if the eyes are closed, so the person has sensory problems which means a problem with dorsal column, medial leminiscus pathway 21:30:35 [butter] yeh i c, the dark room suggest can't use eyes to adjust 21:31:08 [butter] i was thinking this way but confused DC with vestibulo prob, always confuse things 21:31:37 [butter] Where are the cell bodies of spinothalamic tract? 21:31:45 [Step_1] thats ok, better now than in the test 21:32:04 [butter]>[Step_1] yeh sure 21:32:28 [Step_1] VPL 21:32:43 [freaha] butter why dont u tell us about all the cell bodies....the ones u have asked and the ones u r abt to ask....cuz i really dont know 21:33:11 [butter] I just saw these two in my notes right now 21:33:28 [freaha] hmm 21:33:39 [butter] cell body of dorsal column should be in dorsal root ganglia 21:34:04 [butter] cell body of spinalthalamic tract should be in dorsal horn 21:34:42 [butter] cell body of corticospinal tract should be in motor cortex (precentral gyrus?) 21:34:42 [Step_1] thanks 21:35:07 [butter] make sure you can identify the structure when asked about cell bodies, that's what i heard in the lecture 21:36:00 [Step_1] what disease will effect both upper and lower motor neurons? 21:36:46 [butter] amyotrophic lateral sclerosis 21:36:57 [freaha] ALS 21:37:18 [Step_1] ALS (Lou Gerigs Dz) will effect both UPN and LMN....good job butter and freaha 21:38:48 [butter] if barbinski sign is positive on the right, where is the lesion of UMN, left or right? 21:39:40 [Step_1] UMN on the left 21:39:58 [freaha] butter the both sensory tracts have there cell bodies in dorsal root ganglia....check it out 21:40:43 [Step_1] well depends....contralateral if in brainstem or above, but can be ipsi if it is in the spinal cord 21:40:50 [freaha] UMN lesion can be on left if above the 21:40:53 [freaha] rite 21:41:28 [butter]>[Step_1] yeh, can't tell depending on above or below pyramidal decussation 21:42:07 [freaha] wht does syringomylia cause.... 21:42:07 [Step_1] almost had me....had to think that one through a bit 21:42:36 [Step_1] bilateral loss of pain and temp at the level of the lesion 21:43:45 [butter] they point to spinothalamic tract, at that point assume lesion of tract is below cervical spine, the cell body of this tract should be the second neuron, which is dorsal horn 21:44:26 ash enters this room 21:44:39 [Step_1] hi ash 21:44:45 [freaha] yeah rite step 1....cuz it causes a central cavitation...and destroy the crossing over spinothalamic tracts 21:44:48 [ash] hi everyone 21:44:49 [hutals] hi ask 21:45:07 [butter] hi ash 21:45:21 [ash] hi all 21:45:24 [Step_1] infection in caverous sinus gives symptoms of diplopia, eyes pointed nasally, medially strabismus....which CN is effected? 21:45:43 [butter] abducens VI? 21:45:48 [ash] abducent 21:45:57 [freaha] hi ash 21:46:07 [ash] hi 21:46:23 [Step_1] yes, these are signs of deficit in abducens or CN 6, which goes through the cavaernous sinus 21:47:01 [ash] lesion at the base of pons can give what syndrome? 21:47:03 [butter] horner syndrome is always ipsilateral, true or false? 21:47:38 [ash] where corticobulbar and corticospinal tracts are affected 21:47:51 [freaha] true 21:47:58 [Step_1] true? 21:48:39 [ash] insyringomtlia 21:48:46 [ash] of cervical cord 21:48:57 [ash] syringomylia 21:49:49 [ash] alcohol abuse can affect what part of cervix? 21:49:50 [freaha] weber??? 21:49:57 [ash] yes/TO butter 21:50:58 [butter] pontine lesion has medial vs lateral right? 21:51:07 [ash] sorry i meant alcohol abuse can affect what part of cerebellum 21:51:19 [butter] vermis 21:51:26 [freaha] yeah rite 21:51:32 [Step_1]>[ash] ahh, that changes things 21:51:38 [butter] therefore proximal (trunk) is affected, can't keep balance 21:52:12 [Step_1] i agree 21:52:22 [ash] sorry 21:52:38 [Step_1] anterior vermis to be exact 21:53:16 [butter] cerebellar and its peduncle lesion always ipsilateral by the way 21:53:18 [Step_1] no prob....i was scratching my head on that one...lol 21:53:33 [ash] paralysis of upward and downward gaze and noncommunicating hydrocephalus.what syndrome>? 21:53:37 [freaha] ant vermis 21:54:19 [freaha] it starts with a P 21:54:31 [freaha] ......... 21:54:37 [ash] next is a 21:54:58 [ash] parinauds syndrome 21:55:02 [freaha] parinauds 21:55:14 [ash] or dorsal midbrain s. 21:56:27 [hutals] contralat spastic hemiparesis of body, contralat loss of position and vibration sense on body, tongue deviates to lesion side....what syndrom? 21:56:56 [ash] medial medullaryi 21:57:56 [Step_1] medial med....i agree 21:57:57 [freaha] did anyone read about paramedian midbrain syndrome?? 21:58:17 [ash] yes 21:58:19 [hutals] yes, thats right 21:58:33 [ash] it is called benedicts syndrome 21:59:15 [ash] occulomotor n.,dentatothalamic tract and medial lemniscus affected 22:00:19 [freaha] in benedicts ....the dentothalamic fiber lesion causes contralaterl cerebellar dystaxia....does it make sense...???? CONTRALAT. cerebellar 22:00:37 [ash] trigeminal tracts go to which thalamic nucleus? 22:01:32 [butter] do you use kap-lan notes? can't find benedict, remember seeing it 22:01:51 [ash] dentate nucleus projects to contralateral thalamus so it makes sense 22:02:02 [butter] double cross 22:02:11 [butter] equals ipsilateral 22:02:38 [butter] benedicts is also red nucleus syndrome? 22:02:48 [butter] i mean red nucleus lesion 22:03:52 [butter] basal ganglia lesion is to contralateral side 22:04:16 [butter] just so that you know 22:04:51 [hutals] ipsilat limb ataxia, ipsi pain and temp loss in face, contra pain and temp body, vomiting, vertigo, nystagmus away from lesion, horners syndrome, ipsilat paralysus of vocal cord, dysphagia, palate droop. what syndrome? 22:04:58 [ash] trigeminal tract goes to which thalamic nucleus? 22:05:31 [butter] VPN? 22:06:23 [hutals] oops, wasnt supposed to hit sent yet 22:06:58 [butter] medial 22:07:10 [ash] good 22:07:20 [ash] what goes to lateral? 22:07:21 [Step_1] VPM 22:07:34 [butter] pain and tem from face medial, p&t body lateral right? 22:07:55 [ash] right 22:07:58 [freaha] thanks guy si figured it out 22:09:09 [butter] uncus herniation will cause what cranial nerve palsy? 22:09:32 [freaha] last 4 22:09:51 [ash] ipsilateral facial paralysis and medial strabismus what is the site of the lesion? 22:09:55 [Step_1] to help with the vascular lesion syndrome, easiest thing is to figure out from the CN deficit. this will tell you a couple of things. first, which side lesion is on because CN lesions always ipsilat. also will give you idea of level depending on where CN is located 22:10:41 [Step_1] from there, you can match the rest of the symptoms to confirm 22:11:44 [freaha] hutals whts the syndrome 22:12:04 [ash] presbiacusis affects what frequencies of sound? 22:12:25 [hutals] lateral medullary syndrome (aka wallengergs syndrome) 22:13:36 [Step_1]>[ash] i'd like to say medial pons, but sounds more like lat pons?? 22:14:41 [ash]>[Step_1] it is pons but i want the specific site 22:15:04 [butter] what's the afferent and efferent nerve for blink reflex? 22:15:53 [ash] congenital facial diplegia +convergent squint what syndrome? 22:16:56 [butter] yeh i think so 22:17:05 [ash] if then ophthalmic nerve and facial nerve 22:17:09 [freaha] ash is it ...the facial colliculus 22:17:31 [hutals] is it 7 and 9? 22:18:07 [hutals] or 5 and 7 i think 22:18:29 [freaha] 5 and 7 22:18:32 [ash] congenital facial diplegia +convergent squint ,what syndrome? 22:18:50 [butter] what are the nerves that 1. open the eyes 2. close the eyes 3. keep the eyes open? 22:19:28 [freaha] 7 3 7 22:19:36 [freaha] no 3 7 3 22:20:05 [freaha] ash the low frequencies 22:20:18 [ash] yes 22:20:45 [freaha] and tell the difficult syndrome 22:21:14 [freaha] ok 22:21:17 [ash] the syndrome is mobius syndrome 22:21:51 [butter] think it's not on kap-lan notes 22:22:00 [butter] anyway, will check this out later... 22:22:07 [hutals] i couldnt find it in kap lan either?? 22:22:36 [ash] i read h i g h y i e l d 22:22:36 [hutals] where did you get that one from ask? 22:23:11 [ash] it is supposed to be good for anatomy as comparwed to k a p l a n 22:23:15 [hutals] you mean from goljans high yield, or do you mean high yield neuro or anatomy? 22:23:39 [ash] h.h.neuro 22:23:48 [ash] h.y.neuro 22:24:22 [freaha] HY is good...i did a bit of it 22:24:28 [hutals] thanks ash 22:24:39 [Step_1] placidity, excessive excitatory, hypersexuality, hypersatiety, inability to learn, visual and tactile agnosia. what syndrome? 22:24:48 [ash] what lesion of hypothalamus results in poikilothermia? 22:25:28 [ash]>[Step_1] kluver busy s. 22:25:49 [freaha] butter ..so affrent is 3 and effrents r 7 and sym 22:25:57 [hutals] kluver bucy 22:27:33 [ash] ipsilateral anosmia,optic atrophy and contralateral papilloedema.syndrome? 22:27:44 [hutals] posterior nucleus 22:28:18 [Step_1] this is kluver bucy syndrome (ant temporal lobe containing hippocampus and amygdala)....good job ash and hutals 22:28:50 [ash] hippocampus is the most epileptogenic part of cerebrum.why? 22:28:54 [Step_1] agree with hutals, lesion in post nucleus of hypothal results in poikilothermia 22:30:05 [ash] but why do we see that part affected in the first place?? 22:30:43 [ash] no 22:31:48 [Step_1] no to what? its not in the post nucleus? 22:32:01 [ash] ans is the sommers secter in hippocampus is very sensitive to ischemia and therefore epilepsy in this region 22:32:03 [butter] this area is activated constantly, like some routine events? 22:32:14 [ash] no to butters ans 22:32:43 [butter] so it's becoz more prone to ischemic attack? 22:32:46 [butter] i c 22:32:51 [ash] yes 22:33:37 [Step_1] hhhmmm, i didnt even see butters answer. i'm getting a weird connection today 22:33:39 [ash] ipsilateral anosmia,optic atrophy and contralateral papilloedema.syndrome? 22:34:36 [butter] temporal lobe prob? 22:34:36 [Step_1] kennedy syndrome? 22:34:54 [freaha] [ash] what lesion of hypothalamus results in poikilothermia?,,,,asnwer........ 22:35:08 [ash] high dose thiamine therapy,spinal ataxia,optic atrophy and nerve deafness.what syndrome? 22:35:46 [ash]>[Step_1] yes foster kennedy syndrome 22:35:56 [ash]>[Step_1] good 22:37:17 [Step_1] Strachan's syndrome 22:37:23 [ash] wilsons disease affects which nucleus of cns? 22:37:35 [ash]>[Step_1] i am impressed 22:38:25 [hutals] none of this stuff is in kap lan, so now i feel stupid. do we need to study stuff on top of everything in kap for neuro, or is kap enuf? 22:39:31 [freaha] wilson effect basal ganglia 22:39:33 [butter] i watched kap-lan new videos in anatomy and neuro, a lot more shorter than old one 22:39:49 [freaha] I GUESS <a target=new href=http://click.linksynergy.com/fs-bin/click?id=c97WUMRO5hY&offerid=47491.10002441&type=3 &subid=0 >Kaplan</a><IMG border=0 width=1 height=1 src=http://ad.linksynergy.com/fs-bin/show?id=c97WUMRO5hY&bids=47491.10002441&type=3&sub id=0 > IS NOT ENUF 22:39:57 [ash] i suggest h i g h y i e l d series for anatomy 22:40:47 [freaha] i used ridiculously simple and bit of HY bit of <a target=new href=http://click.linksynergy.com/fs-bin/click?id=c97WUMRO5hY&offerid=47491.10002441&type=3 &subid=0 >Kaplan</a><IMG border=0 width=1 height=1 src=http://ad.linksynergy.com/fs-bin/show?id=c97WUMRO5hY&bids=47491.10002441&type=3&sub id=0 > 22:41:03 [hutals] i thought i knew alot by learning kap lan neuro past few days, but i havent seen alot of the questions ash is asking 22:41:18 [butter] i feel same 22:41:38 [freaha] me too feel bad 22:41:46 [Step_1] freaha, you cant type words like kap lan, amaxon, first aid, etc or you will get that link 22:41:55 [butter] in the new video they stress that only 3 long tracts are required to know, as to current trend, then brain stem lesion important too 22:42:15 [freaha] ok ash tell all the answers.... 22:42:30 [freaha] ok i get it 22:42:30 [ash] come on guys you are doing well 22:42:53 [ash] please dont feel bad .that was not my intention 22:43:14 [hutals] i agree, kap is not as in depth because it covers only the really high yield stuff. 22:43:30 [freaha] no its good tht we know wht to do....need to study more of neuro 22:43:50 [ash] 22:43:54 [hutals] we know ash that you did not have bad intentions. i think we just studied different sources so hard to answer your questions. 22:44:20 [butter] after all we are taking same exam 22:44:41 [freaha] [ash] wilsons disease affects which nucleus of cns? 22:44:48 [hutals] just hope you dont think we didnt put in the effort to study for the session or something because i put alot into it, but its not showing 22:45:06 [freaha] guys dont get down......lets studyyyyyy and learn more and more 22:45:33 [hutals] i agree, i'm learning alot from ash and the rest of you....lets move on 22:45:38 [freaha] so shalll we ......... 22:45:56 [Step_1] ok, where were we? 22:47:04 [ash] pt.has topographic memory loss,anosognosia,construction apraxia,dressing apraxia,what part of the brain is affected? 22:47:19 [freaha] basal ganglia.... 22:47:43 [freaha] i dont know abt caudate 22:48:38 [freaha] [ash] high dose thiamine therapy,spinal ataxia,optic atrophy and nerve deafness.what syndrome..answer plz 22:49:05 [hutals] middle cerebral artery stroke? 22:49:36 [freaha] Strachan's syndrome.....wht is this syndrome 22:49:50 [freaha] ok 22:51:28 [hutals] basal ganglia, and the posterior and anterior internal capsule? 22:52:10 [butter] what's the wilson answer, ash? 22:53:04 [ash] now you guys ask some questions 22:53:14 Pascal enters this room 22:53:25 [freaha] IT IS THE INFERIOR PARIETAL LOBE OF NON DOMINANT HEMISPHERE 22:53:35 [hutals] your asking some good questions ash, please continue 22:53:48 [Step_1] hi pascal 22:53:48 [freaha] 22:54:03 [freaha] HI PAS 22:54:11 [ash] what test is used to test cerebral dominance? 22:54:19 [ash] hi pascal 22:54:35 crusher enters this room 22:54:53 [crusher] hi everyone 22:54:58 [freaha] SHOW ME WHICH HAND U WRITE WITH...ASH SO I CAN TEST U 22:55:11 [freaha] AM I RITE 22:55:18 [Step_1] what is the blood supply to the anterior limb of the internal capsule? question actually had a pic of the three limbs and pointed to an infarction and asked which vessel caused this 22:55:29 [Step_1] hi crusher 22:55:34 [freaha] HI CRUSHER 22:56:02 [ash] hi crusher 22:56:13 [butter] hi crusher 22:56:30 [freaha] HOW DO U CHECK FOR CEREBRAL DOMINENCE 22:57:12 [butter]>[Step_1] ante limb ACA, rest of it MCA 22:58:17 [Step_1] excellent butter! 22:58:53 [butter]>[Step_1] yeh.....just looked and copied 22:59:29 [ash]>[Step_1] antwerior choroidal artery? 22:59:41 [butter] hypertensive pt wake up in the morning and hd pure motor defect, what's wrong? 23:00:27 [crusher] stroke 23:00:50 [butter] sorry, i mean what part of brain is affected? 23:00:54 [ash] patienthas right and left confusion,finger agnosia,dyslexia,dyscalculia,contralat.hemianopia. what syndrome?site? 23:01:23 [Step_1] corticospinal tract i guess with info given 23:02:18 [crusher] parital lobe ..motor area. 23:03:18 [butter] lateral striate branches of MCOA supply posterior limb 23:04:01 [ash]>[Step_1] is ant.choroidal artery right for ant.limb? 23:04:20 [butter] MCA, not MCOA 23:04:58 [butter] actually post limb that close to genu 23:05:49 [ash] guys what about my syndrome?some ans? 23:07:16 [ash] gerstmanns syndrome involves inferior perietal lobule of dominant hemisphere 23:07:44 [butter] hmm i should know this one 23:07:46 [Step_1]>[ash] it was the ant cerebral artery as the main blood supply. not sure about branches. 23:08:04 [freaha] IN HYPERTENSION...THERE CLD B THROMBOEMBOLISM..OR INTRACEREBRAL BLEED... 23:09:03 [ash]>[Step_1] yes the branches are medial striate which supply the ant.limb 23:09:30 [Step_1]>[ash] thanks 23:09:55 [Step_1] maybe we should do some histo since our time is running out. what do you think? 23:10:03 [butter] ok 23:10:26 [butter] in cardiac muscle, what binds to calcium? 23:11:36 [hutals] troponin? 23:12:22 [freaha] Step_1] what is the blood supply to the anterior limb of the internal capsule? question actually had a pic of the three limbs and pointed to an infarction and asked which vessel caused this...ANSWER PLZ 23:13:33 [ash] ant.cerebral a.(medial striate branches) for ant. limb 23:13:42 [freaha] TROPONIN 23:14:19 [ash] middle cerebral a.(lat.striate branches)for posterior limb 23:14:26 [Step_1] the choices gave anterial cerebral , middle cerebral, post cerebral, and a couple of others. the answer was ant cerebral, but ask pointed out that the branches are the medial striate. 23:15:03 [ash] also ant.choroidal a. which is a branch of internal carotid a. for post.limb 23:15:27 [Step_1] i agree that ca binds to troponin 23:15:45 [butter]>[Step_1] troponin C 23:15:50 [butter] remember this point 23:16:14 [Step_1] thanks butter 23:16:32 [freaha] I DONT KNOW MORE ABT mr TROPONIN 23:17:25 [Step_1] brush border seen in what part of kidney? 23:17:54 [butter] proximal convoluted tubule 23:18:06 [hutals] prox tubule 23:18:13 [crusher] prox tubule.most reabsorption there 23:18:23 [butter] brush border for better absorption, so PCT and small intestine absorb a lot 23:18:37 [Step_1] yep, proximal convoluted tubule. great job! 23:18:56 [ash] also straight portion of loop of henle 23:19:51 [ash] where do you find principle cells and intercalated cells? 23:21:46 [ash] ok guys i have to go ,it was a good experience,thankyou all. 23:21:57 [Step_1] prox tubule 23:22:07 [butter] bye ash 23:22:09 [ash] goodnite and happy studying 23:22:35 [freaha] BYE AND THANK U 23:22:53 [hutals] thanks ask 23:23:02 [Step_1] bye ash 23:23:27 [ash] bye 23:24:03 [Step_1] what cells secrete HCL? 23:25:03 [hutals] parietal cells 23:25:06 [freaha] PARIETAL 23:25:24 [Step_1] parietal cells secrete HCL...good job 23:25:42 [Step_1] what about secretion of pepsinogen? 23:25:58 [butter] chief cells 23:26:08 [hutals] chief cells 23:26:27 [freaha] WHT MAKES THE BLOOD -AIR BARRIER 23:26:32 [Step_1] yep, chief cells. great! 23:28:25 [Step_1] type 1 cells, basal lamina and endothelial cells 23:28:56 [hutals] agree 23:29:06 [freaha] RITE 23:29:19 [Step_1] of the salivary glands which one has only serous? which one has both but mostly serous? and which one has both but mostly mucus? 23:29:42 [hutals] serous is the parotid 23:30:22 [freaha] MOSTLY SEROUS IS SUBMANDIBULAR 23:30:44 [freaha] MOSTLY MUCOUS IS SUB LINGUAL 23:30:59 [hutals] agree 23:31:16 [Step_1] parotid for only serous, submandibular for both but mostly serous, and sublingual for both but mostly mucus ( last one was a q.bank question)....great again! 23:31:18 [freaha] WHICH TYPE OF ANTIBODY IS MADE BE PEYERS PATCH 23:31:34 [butter] IgA? 23:32:30 [freaha] YES RITE IgA 23:34:09 [Step_1] which is more abundant....type 1 or 2 pneupomocytes? 23:34:35 [freaha] 1 23:34:54 [butter] 1 23:35:30 [Step_1] type 1 make up 95% of alveolar surface and very important for gas exchange...excellent....even though i misspelled 23:36:29 [freaha] MITOCHONDRAL DNA COMES FROM MOM OR DAD??? 23:36:48 [Step_1] mom 23:37:01 [hutals] mom 23:37:30 [freaha] YUP.... 23:37:44 [Step_1] what type of collegen for basement membrane? 23:38:08 [freaha] 4 23:38:34 [freaha] WHT IS DEFICINT IN I -CELL DISEASE 23:38:45 [Step_1] yep 4 for BM.......what about for bone? and for cartilage? 23:38:59 [Step_1] mannose phosphate 23:40:27 [freaha] ONE IN bONE....TWO IN carTWOLAGE......FOUR....UNDER THE FLOOR...(basement) 23:41:16 [Step_1] UDP-N-acetylglucoseamine-1-phosphotransferase 23:41:35 Gianna enters this room 23:41:39 [Step_1] very good freaha....i like that 23:41:44 [Step_1] 23:41:53 [Step_1] hi gianna 23:41:55 crusher enters this room 23:42:06 [Gianna] Hi 23:42:10 [hutals] hi gianna. welcome back crusher 23:42:14 [freaha] RITE...IT IS THIS ENZYME.... 23:42:33 [Gianna] I just came by to see what's going on, i am new 23:43:01 [Gianna] are you doing anatomy I guess? 23:43:22 [freaha] YES WE R ... 23:43:53 [Step_1] yes. anatomy all week, so split into two sessions. tonight is neuro (already finished) and histo. sat is embryo and gross 23:44:39 [freaha] WHERE DO U FIND...dynein 23:44:43 [Step_1] but we end around 12 pm eastern, so almost done for tonight . anything specific you wanted to go over? 23:45:44 [Step_1] cilia, but absent in kartageners syndrome 23:46:44 [freaha] ITS IN CILIA.... 23:47:33 [Step_1] in follicular development, what stains pink with PAS stain? 23:48:22 [freaha] AND IN RETROGRAD.... 23:49:19 [Step_1] hunt: acrosome reaction occurs when sperm reaches the part that stains pink with PAS stain 23:49:45 [freaha] ZONA PELLUCIDA...AS IT IS RICH IN POLYSACCHARIDE 23:50:09 [Step_1] both were q.bank questions....the zona pellucida stains pink with PAS stain....good job 23:50:27 [Step_1] AND IN RETROGRAD....didnt get the question 23:51:04 SMS enters this room 23:51:31 [Step_1]>[Gianna] we've covered just about everything, is there anything specific you wanted to discuss? 23:51:37 [Step_1] hi sms 23:51:46 [SMS] hi 23:51:46 [freaha] DYNEIN....WORKS IN RETROGRADE AXONAL FLOW...AND IT WORKS IN CILIA ... 23:51:54 [freaha] HIII 23:52:00 [freaha] HOW DEEE 23:52:03 [Step_1] ah....makes sense....thanks 23:53:26 [Step_1] anything else anyone want to discuss before we call it a night? 23:54:03 [hutals] nope, i'm good. i learned alot tonight 23:54:51 [butter] planning to get H Y neuro 23:55:01 [freaha] WHT R THE pH DIFFENECES IN ESTROGENIC...AND POST ESTROGENIC PHASE OF VAGINAL FLIUD 23:55:13 [SMS] when is neuroanatomy chat? 23:55:37 [hutals] we covered neuro earlier tonight 23:55:53 [SMS] oh 23:55:59 [freaha] IT WAS 3 HRS EARLIER 23:56:17 [SMS] what are the subjects for friday? 23:56:51 [hutals] just when i thought i knew it all, i found out that HY neuro is very detailed compared to kap lan 23:57:41 [Step_1] its actually on sat at 9 pm eastern and will cover embryo and gross anatomy 23:58:00 [freaha] YEAH BUT NEUROANATOMY IS ONE THING ...MOSTLY ASKED IN ANATOMY....SO GIVE IT MORE TIME... 23:58:49 [SMS] thanks 23:59:02 [hutals] thats true...i'm just wondering if i should buy hy neuro or stick with kap lan? 23:59:15 [freaha] IN ESTROGENIC PHASE pH IS LOW...IN POSTESTROGENIC PHASE pH IS HIGH...SO MORE CHANCES OF INFECTION IN THE LATTER.... 23:59:29 [Step_1] no prob sms. hope to see you on sat. 00:00:03 [freaha] OKAY EVERYONE....I LL BE GOING NOW... 00:00:11 [Step_1] oh, sorry freaha for not answering....forgot about the question 00:00:25 [Step_1] ok, so i guess i'll say good night, thanks for chatting, and see you all on sat for the rest of anatomy 00:00:31 [freaha] SEE U ON SAT...I WAS A VERY GOOD EXPERIENCE... 00:00:39 [freaha] BYEEEEEEEEEE 00:00:49 [hutals] goodnight everyone and thanks 00:01:20 [butter] good luck step 1 00:01:56 tau enters this room 00:02:01 [Step_1] thanks butter |
![]() |
| Thread Tools | |
| Display Modes | |
|
|
|
|
||||
| Thread | Thread Starter | Forum | Replies | Last Post |
| chat transcript - histology | Anonymous | USMLE Step 1 Forum | 0 | 11-16-2004 10:39 PM |
| Chat transcript - Anatomy (Embryo and Histo) | Anonymous | USMLE Step 1 Forum | 0 | 09-14-2004 10:42 PM |
| chat transcript - behavioral science (sleep, dsm, cortical f | Anonymous | USMLE Step 1 Forum | 0 | 07-17-2004 11:55 PM |
| chat transcipt - anatomy (embryo and histo) | Anonymous | USMLE Step 1 Forum | 1 | 05-27-2004 02:54 PM |
| transciption of usmle chat | Anonymous | USMLE Step 1 Forum | 1 | 05-14-2003 08:58 PM |
International Foreign and Caribbean medical schools,
ValueMD provides information on medical education from premed to residency