[bioche2006] which type of glomerular nephritis seen in AIDS
[usmle.doc] focal ssegmental glomerulo sclerosis
[bioche2006] MCC OF NEPHROTIC SYNDROME IN ADULTS
[usmle.doc] membranous glomerulonephritis
[bioche2006] good
[bioche2006] in kids?
[usmle.doc] minimal change disease in children
[bioche2006] good
niks has left the chat.
[bioche2006] in sle?
[usmle.doc] five types of renal involvement is there right
[perhaps] diffuse proliferative MC?
[perhaps] no diffuse membranous, sorry
[mahitha] diffuse proli gl
[bioche2006] everyone
[usmle.doc] ok most common is focal prolifer??
[mahitha] gl.nephritis
[bioche2006] which one most common i am confused too
[mahitha] diffuse
[usmle.doc] it si diffuse sorry
[bioche2006] ok
niks has left the chat.
[bioche2006] which type of kidnye stone most common
[bioche2006] ?
[usmle.doc] calcium oxalte?
[bioche2006] rt
[bioche2006] if u see wbc rbc in urine where infection
[usmle.doc] ok
[usmle.doc] k
[perhaps] renal
[niks] hello everyone...did the chat begin?
[bioche2006] wrong
[perhaps] if you see wbc cast
[bioche2006] rt if cast then renal involvement
[perhaps] hi niks
[bioche2006] but as such rbc wbc bladder
[perhaps] yes
[niks] hi perhaps!
[bioche2006] allrt guys i idnt come prepared i asked whatever came to mind
[bioche2006] u can begin
[niks] joe didnt turn up today?
[usmle.doc] subendothelial deposits are seen in which type of glomerulonephritis
[niks] SLE
[bioche2006] sle
[perhaps] he showed up, but his computer is running out of battery, so he went to take care of that. At 8:25 he should be back
[usmle.doc] and
[perhaps] MPGN
[perhaps] amyloid
[usmle.doc] yes mpgn type 1
saimakm has joined subroom: USMLE_Step_1
[niks] oh...u mean 9:25
[perhaps] yes, sorry niks
[niks] haha...ok
[perhaps] which GN is associated with hepatitis B?
[junglemits] topic today?
[perhaps] renal
[saimakm] mengoprolifera
[bioche2006] mmpg
[perhaps] yes goljan said it is membraneous
[usmle.doc] membranous glomerulonephrits
[perhaps] yes
[niks] yes its membranous
[perhaps] which other disease with renal involvement and vasculitis is alos associated with hepatitis B?
[perhaps] also
[bioche2006] pan
[perhaps] yes
[junglemits] panca
[niks] PAN
[usmle.doc] poly artetitis nodosa
[saimakm] oh yes mmpg is c
[perhaps] right that is HCV
[saimakm] thanks
[niks] which renal disease is associated with faom cells in tubular epithelium
[saimakm] alports
[usmle.doc] alpots
[niks] yea..
[niks] which seen most commonly in IV drug abusers
[perhaps] focal segmental
[saimakm] focal glomirular
[bioche2006] fsgc
[niks] yea...
[junglemits] and aids
[bioche2006] rt
[bioche2006] same
[niks] it has poor resopnse too steroids too
[niks] ok which is the mc cause of nephrotic in adults?
[bioche2006] membranous
[saimakm] memb
[niks] yea...
[niks] what is tertiary hyperparathyroidsm?
[bioche2006] after treamtnet of seconday hyper pth due to hyperplasia stil it secretes incresed amount of pth
[usmle.doc] when the parathyroids become autonomous due to long standing secondary hyperparathyroidsm
[niks] yea...its hypercacemia which is not able to supress the PTH...both remain high
[bioche2006] so incresed ca decresed phophorus adn incresed pth
[bioche2006] rt
[niks] mannitol works at which site of renal tubule...I mean which section?
[bioche2006] pct
[usmle.doc] proximal
[perhaps] and thick asending, and CT?
[niks] u know what I read...
[saimakm] osmotic so thick asendin
[bioche2006] it does but most affect is at pct where it prevent most of reabsorption
[niks] i read that it works on the thin descending limb...I'll tell u how
[bioche2006] thick ascending is any way impermeable to water
[niks] the thin descending limb is impermeable to solutes but permeable to water...
[bioche2006] rt
[saimakm] make sence
[niks] and is a site of water reabsorption
[bioche2006] but if niks u remebr 60 percen to f absorption of things occurs in pct
[saimakm] thanks
[bioche2006] rest all doesnto make much diference
[niks] mannitol and other osmotic agents work by preventing water reabsorption at this site
[bioche2006] areu sure niks iti s thin decending ok
[niks] I read in physio q bank...
[bioche2006] then i wll agree i read it as pct
[bioche2006] allright
[bioche2006] thnaks niks
[niks] yes in PCT water is reabsorbed but thats isoosmotic
[niks] reabsorption
[niks] in thin descen only water reabs no solute
madalasa has left the chat.
[niks] perhaps...do u agree?
[perhaps] I have to look up
[niks] ok...any way it was in the physio q bank...
dr_ashima has left the chat.
[usmle.doc] ok thank u niks
[niks] ure welcome...
[perhaps] Hi, in kaplan pharma, it says PCT, thin descending and CT
[niks] a person has pain radiating in groin and hematuria...what mightbe the diagnosis...
[perhaps] kidney stone
[usmle.doc] renal calculi
[saimakm] stones
[niks] ok..perhaps....I made anote of it...thanks
[perhaps] welcome
[bioche2006] rt niks i checked in kaplan q bank it says descedning limb
[bioche2006] good one niks
[niks] yes its a kidney stone...wont it be more specifically a stone in the ureter?
[usmle.doc] yes niks
[niks] u mean thin descending limb as I said bioche?
[bioche2006] yeah niks mannitol affects descending limb
[niks] coz stone in the ureter can give rise to colic...and stone in kidney gives the costvertebral angle tenderness
usmle.doc has left the chat.
[bioche2006] rt
[niks] thank you bioche...
[bioche2006] u r w
[niks] a patient has APKD..and dies suddenly...what might be the cause?
[bioche2006] sub arachnoid hge
[usmle.doc] rupture of bery anersyms
[usmle.doc] berry aneurysm
[niks] yep....where does the hemorrage occur?
[bioche2006] yeah berry aneurysm
[perhaps] subarachnoid
[usmle.doc] into subarachnoid space
[niks] yep...
[usmle.doc] genes regulating apkd are
[niks] what is the most common cause of calcium oxalate stones...
[bioche2006] dominant
[niks] oops..
[niks] dominant
[saimakm] 16
[bioche2006] hyper pth
[usmle.doc] yes dominant and they apkd 1 on 16 and apkd 2 on chr 4
[niks] nope...
[niks] not hyperPTH
[bioche2006] dont know
[saimakm] diet
[niks] thank u usmle_doc
[bioche2006] loopdiurteics
[niks] its idiopathic hypercalceuria...cause is not known
[bioche2006] oh
[usmle.doc] ok
[saimakm] ok
[niks] antiglomerular basement membrane antibodies damage both GBM and pulmonary alveoli...whats the diagnosis?
[bioche2006] good pasteur
[josephmedman] hey guys
[saimakm] good pas
[bioche2006] hi joseph
[perhaps] hi
junglemits has left the chat.
[josephmedman] what have you guys gotten to?
[usmle.doc] good pasteurs
[niks] yep its goodpasturs
[josephmedman] ok
[niks] just random quest...
[niks] nice discussion...I shall send that part to u
[josephmedman] ok cool
[josephmedman] should i throw my order at you?
[josephmedman] from the start?
[niks] sure!
[josephmedman] or start from goodpastures and move forward?
[saimakm] why not
[josephmedman] ok cool..
[niks] oh we had no order...haha
[josephmedman] im gonna go quick because we have a lot to cover
[niks] yep!
[josephmedman] what is the 1st metabolic abnormality in renal disease?
[niks] proteinuria
[josephmedman] proteinuria
[josephmedman] what is the 1st indication of diabetic nephropathy?
[bioche2006] microprotin
[josephmedman] microalbuminuria
[bioche2006] rt
[usmle.doc] microscopic proteinuria
[josephmedman] what is the first thing that happens in renal failure?
[niks] oligura
[josephmedman] loss of ability to concentrate urine..
[bioche2006] rt
[niks] oh...
[bioche2006] not necessary
[josephmedman] neha jen or anyone..if you guys want to throw anything at anytime in please be my guest
[niks] means dilute urine...
[niks] sure joe...thanks alot
[bioche2006] that depends upon type of renal flaiure
[josephmedman] umm..well im jut going by goljan notes
[bioche2006] ok
[niks] yep acute tubular necrosis first gives oliguria..and then polyuria on improvewment
[bioche2006] controversial topic next q
dr_ashima has left the chat.
[josephmedman] ok
[josephmedman] what is it when you see rbc casts in urine?
[bioche2006] renal parenchyma inolv
[niks] nephritic...
[josephmedman] glomerulonephritis
[saimakm] nephritic
[niks] kidney involverment
[josephmedman] what about WBC's in urine?
[bioche2006] nephritic to be
[josephmedman] sign of infection
[saimakm] pyuria
[bioche2006] wbc can even come from bladder infection
[niks] viral infection...
[niks] or UTI
[josephmedman] what is the MC stone in urine?
[bioche2006] rt
[bioche2006] calcium oxalate
[niks] calcium oxalate
[saimakm] calsium oxa
[josephmedman] what is the MC site for acute tubular necrosis?
[perhaps] PCT
[niks] PCT?
[bioche2006] no
[josephmedman] what part?
[saimakm] pct
[niks] the site where metabolic activity is highest is PCT
[josephmedman] medullar section because it receives only 10 percent of the blood supply
[bioche2006] i thouhg loop of henle
[perhaps] ohh, yes
[josephmedman] 90 percent is in cortex
[bioche2006] rt
[niks] oh thanks
[josephmedman] since only 10 percent of blood supply
[josephmedman] more susceptible to injry
[perhaps] thanks, that's right
[josephmedman] what is the MC glomerular disease?
[niks] maybe...membranous Gn
[josephmedman] no
[josephmedman] IgA glomerulonephritis
[niks] oh it might be diabetic glomerulopathy?
[niks] oh ok...
[bioche2006] goodone
[josephmedman] what is the MC congenital disorder of the kidney?
[saimakm] ok
[niks] horseshoe kn
[josephmedman] good
[bioche2006] gree
[josephmedman] and what congential genetic disorder is it associated with?
[perhaps] turner
[josephmedman] good
[josephmedman] what is turners?
[bioche2006] xo
[ppliutcm] 45x0
[perhaps] 45 xo
[josephmedman] where is the kidney trapped behing is horshoe kidney?
[perhaps] superior messentaric artery
[josephmedman] no
[usmle.doc] below inf mes artery
[saimakm] inf
[josephmedman] good
[josephmedman] inferior
[perhaps] right
[josephmedman] what type of trait is juvenile polycystic kidney disease?
[bioche2006] recessive
dr_ashima has left the chat.
[niks] behind in the retroperitoneum below the bifurcation of aorta
[josephmedman] what do you see in the mother
[bioche2006] recessive
[josephmedman] and what characteristics do you see in the bady
[perhaps] oligoamnois
[josephmedman] niks what is below the bifurcation of aorta?
[bioche2006] rt
[perhaps] potter in baby
[bioche2006] agree oligo
[niks] I thought the horse shoe kn is trapped there
[josephmedman] hypoplasia of lungs is an impt. consequence of oligohydramnios
[bioche2006] rt
[josephmedman] no..thats something else i think
[josephmedman] i cant think of it right now though..
[niks] ok...
[josephmedman] what chromosome mutation is APKD?
[niks] we check that out...later
[bioche2006] dominant
[niks] 16
[josephmedman] what chromosome?
[josephmedman] good
[josephmedman] what is associated with it?
[bioche2006] berry
[josephmedman] board favorite
[bioche2006] aneurysm
[josephmedman] great
[niks] berry
[josephmedman] what heart condition associated?
[perhaps] MVP
[josephmedman] good
[niks] MVP
[bioche2006] what is it
[saimakm] mvp
[josephmedman] what is the MCC of hematochezia?
[bioche2006] what is mvp
[perhaps] mitral valve prolapse
[josephmedman] mitral valve prolapse
[usmle.doc] =diverticulosis?
[bioche2006] oh thanks
[josephmedman] good
[saimakm] diverticulitis
[josephmedman] if something ends in
[josephmedman] "itis"
[josephmedman] what type of hypersensitivity reaction is it?
[bioche2006] type 3
[niks] 1
[josephmedman] type 3
[niks] oh..
[niks] sorry
[josephmedman] if on IF ther eis a linear patter what does that mean?
[bioche2006] good pasteur
[niks] GBM ab-good pasteur
[josephmedman] means that antiBM antibodies are the cause..
[bioche2006] rt
[josephmedman] if there is a granular patter what does that mean?
[bioche2006] streptococala
[niks] immune complexes
[josephmedman] it means immune complexes are deposited
[bioche2006] immune complx
[saimakm] post strep
[josephmedman] good niks
[usmle.doc] post streptococcal
[niks] PSCG
[josephmedman] heparin sulfate what is it?
[niks] thanks
[bioche2006] basement menbrane protein
[josephmedman] keeps albumin out of the BM
[saimakm] basement memb
[bioche2006] rt
[niks] anion on gbm
[niks] prevents albumin loss
[bioche2006] becz od negative carge
[josephmedman] remember with subepithelial it is very small and it can go thru the BM
[bioche2006] rt
[josephmedman] and with sub endothelial it is too big and it cannot go thru the BM
[bioche2006] sle
[josephmedman] what is the MC mechanism prod. acute glom nephritis?
[niks] oh..
[bioche2006] post strep
[usmle.doc] immune complex deposition
[niks] post strep
[josephmedman] type 3 hypersensitivity deposits..(eg. SLE)
[josephmedman] when do you see wire looping of the capillaries?
[josephmedman] and everyone please look at this pic on webpath..
[saimakm] sle
[josephmedman] it is very high yield..
[usmle.doc] in sle
[josephmedman] what is the MCC of death in SLE?
[erum] sle
[bioche2006] rf
[josephmedman] infection..
[bioche2006] oh
[bioche2006] thanks
[josephmedman] what type of antibodies will you see?
[bioche2006] anti dna
[niks] anti DNA
[ppliutcm] ana
[josephmedman] anti-DNA
[bioche2006] in drug induce anti histone
[erum] anti dna
[josephmedman] which is the worst type of glomerulonephritis?
[josephmedman] oh ok
[saimakm] focal seg
[perhaps] crescent
[bioche2006] fs
[niks] rapidly progessive
[erum] fs
[usmle.doc] fsgs
[josephmedman] crescentic
[bioche2006] rt
[josephmedman] within 3 months you will go into renal failure
[josephmedman] and die
[bioche2006] rt
[niks] yep...
[josephmedman] in which disease do you see the presence of cholesterol in the urine?
[bioche2006] nephrotic
[josephmedman] ok..
[erum] neph
[bioche2006] lipoid
[niks] fat bodies in nephrotic
[bioche2006] i mean minimal
[josephmedman] what is the MC disease causeing nephrotic in children?
[bioche2006] minimal
[usmle.doc] minimal change
[niks] lipod nephrosis
[saimakm] nil
[josephmedman] good you have a loss of negative charge
[josephmedman] what do ALL patients with nephrotic syndrome have?
[bioche2006] rt effacemtn of foot process
[niks] whats the pathological mechanism in this...
[josephmedman] fusion of podocytes and hypercholesterolemia
[niks] in minimal?
[josephmedman] in what niks?
[niks] in lipod nephrosis
[josephmedman] t cell immune reaction against viceral epithelial cells niks
[niks] yep...
[josephmedman] for minimal change
[bioche2006] gog done joseph about all
[bioche2006] ALL
[josephmedman] ?
[niks] yes joe..right
[bioche2006] i mean good question on ALL
[josephmedman] have fusion of podocytes and hypercholesterolemia
[josephmedman] oh ok..
[josephmedman] haha..
[josephmedman] what is the MC nephropathy in HIV and IV heroine abuse?
[josephmedman] this is another fav.
[niks] FSGN
[bioche2006] fsgs
[saimakm] focal seg
[josephmedman] great
[josephmedman] are there electron deposits?
[bioche2006] no crescents
[saimakm] no
[josephmedman] NO
[niks] no dposits
[josephmedman] what can captopril cause?
[bioche2006] renal faliure
[josephmedman] diffuse membranous
[bioche2006] oh
[josephmedman] which one is associated with HBV?
[niks] renal artry stenosis
[perhaps] MPGN1
[niks] oh ok
[saimakm] memb
[josephmedman] no
[perhaps] no
[josephmedman] membranous yes
[niks] ok joe...
[josephmedman] which one is membranoprolif assoc with?
[saimakm] hcv
[perhaps] HCV
[josephmedman] good
[josephmedman] what is the MCC of chronic renal failure in US?
[perhaps] diabetes
[josephmedman] DM
[bioche2006] diabetes
[josephmedman] what happens to the BM?
[bioche2006] thickened
[josephmedman] non-enzymatic glycosylation
[bioche2006] with abnomral glycosylaton
[josephmedman] what happens to the GFR in this?
[bioche2006] decresed
[josephmedman] increased because of narrow lumen..
[niks] also sorbitol damage
[saimakm] increased
[bioche2006] oh
[josephmedman] you have an increased pressure
[niks] increased
[bioche2006] tx
[josephmedman] what type of collagen assoc with this?
[perhaps] 4
[bioche2006] type 4
[niks] 4
[josephmedman] what heridatry defect is the defect in synthesis of type 4 collagen?
[bioche2006] oi
[perhaps] ED type 3?
[niks] ehrler danlos
[josephmedman] alport.what type of disease is this?
[richtian] alport
[saimakm] osteo imper
[perhaps] Autosomal dominant
[perhaps] no
[josephmedman] SXR
[bioche2006] wait joseph answer
[perhaps] SXD
[josephmedman] no no
[josephmedman] SXD
[perhaps] yes
[bioche2006] for defectin type 4 collagen
[josephmedman] SXD
[niks] sex linked dominant
[josephmedman] sorry
[josephmedman] comp is flipping
[josephmedman] what is fabry disease?
[niks] thats alport bioche
[bioche2006] x linked
[bioche2006] oh tx
[niks] defect in alpha4 chin of typer 4 collagen
[bioche2006] fabrys only x likned
[niks] sorry alpha 5 chain
[josephmedman] sxr lysosomal strorage disease..de. in alpha galactosidase..EM..lamellar inclusions..
[bioche2006] thnaks nik
[josephmedman] what is prerenal azotemia?
[bioche2006] incresed urea compared to creatinine
[niks] due to decresed perfusion of kn
[josephmedman] what is CO?
[josephmedman] in prerenal?
[josephmedman] and what is GFR?
[bioche2006] decresed
[niks] decresed
[josephmedman] and explain why
[bioche2006] gfr decresed
[erum] dec dec
[saimakm] co dec
[josephmedman] and what is BUN and explain why?
[bioche2006] beocz prerenal means decresed bllod flow to kidney hence gfr also decresed
[erum] inc
[josephmedman] why erum..
[bioche2006] blood urea nitrogen incresed
[niks] BUN increased
[perhaps] BUN is reabsorbed more now
[josephmedman] more time to reabsorv urea
[bioche2006] rt
[niks] yep
[erum] slow gfr
[josephmedman] what is acute tubular necrosis?
mahitha has left the chat.
[erum] inc bun absorption
[niks] due toischemia
[saimakm] creatinine is not much absorbed in compare to urea
[erum] dis proportionate
[josephmedman] no
[bioche2006] there r types and in ATN failure of urine to concetrate occurs
[josephmedman] BUN and creatinine are equally affected with tubular dysfunction
[josephmedman] in acute tubular necrosis
[bioche2006] yeah
[saimakm] isch
[erum] equal in atn
[bioche2006] prerenal urea moe incresed compared to cretatinin and in renal both equally incresed
mahitha has left the chat.
[josephmedman] ok..
[niks] yes bioche right
[josephmedman] i have a question if anyone can answer
[bioche2006] tx
[josephmedman] in ischemic ATN there is loss of the Na in the urine
[bioche2006] rt
[josephmedman] can someone relate that to the loss of the ability of the kidney to concentrate urine in renal failure?
[josephmedman] anyone know the mechanisms?
[bioche2006] yeah becoz loops of henle gone hence the medullary osmotic conc gradient gone hence
[niks] in the beginning when there is oliguria there is Na retention...then diuressis results losing na...
[josephmedman] ok..
[niks] this is what I can recall...I shall double check this
[erum] dec in medulary osmotic conc
[josephmedman] so ischemic ATN is the latter half then?
[bioche2006] yeah we get conc urine only if medullary conc gradient maintained
[perhaps] es
[perhaps] yes
[bioche2006] thats why we say max conc we can do is till1800
[bioche2006] bec thats max medullary conc gradiant
[niks] I though 1200
[saimakm] rt
[erum] after pct reabsorption is dependent on medulary conc
[bioche2006] may be 1200 iam not sure i a giving relevance thansk niks fro correctign me
[josephmedman] wait..but how does ischemic ATN cause that maintenence of the concentration gradient
[bioche2006] no it is not there
[josephmedman] like hwo do we lose Na in ATN is my question
[perhaps] they can't reabsorb, can't concentrate either i think
[niks] hey..
[bioche2006] rt
[perhaps] lose more water than Na?
[niks] the GFR is intact in ATN...
[erum] loss of -ve charge on memb
[perhaps] yse
[niks] so na is filtered...but no reabsorption
[perhaps] yes
[josephmedman] ahhh..ok...
[niks] UNa>40
[josephmedman] what is MCC of ATN?
[josephmedman] thanks guys..
[bioche2006] hold on wait , in ascending loop of henle we reabsorb 2 na cl into meduallry interstitium, which then pulls water from decending limb, if ATN occurs then we cannot absorb thru that pum and more and more water and sodium lost
[niks] and FENa>1
[niks] ischemia is thew MCC
[saimakm] ischemia
[josephmedman] no
[bioche2006] drugs
[josephmedman] no
[bioche2006] nsaids
drnm has left the chat.
[bioche2006] then
[josephmedman] ooops
[josephmedman] i meant what is the MCCOD
[bioche2006] didnt get you
richtian has left the chat.
[niks] whats MCCOD...
[josephmedman] most common cause of death
[perhaps] bioche2006 thanks nice explaination
[josephmedman] thats bioch i understand perfectly now..
[niks] oh...sorry...thanks
[bioche2006] u r w
[niks] its the dehydration I guess...
[josephmedman] infection is the answer
[bioche2006] hyperk
[josephmedman] what is the most common in nephrotoxic ATN?
[niks] thanks bioche
[niks] oh ok...
[bioche2006] u r w
[josephmedman] aminoglycosides
[saimakm] aminoglycosides
[josephmedman] what happens to estradiole in klinefelters?
[bioche2006] incresed
[josephmedman] where does fetal testosterone develop?
[niks] increased...
[saimakm] increased
[niks] in adrenals
[josephmedman] no
[bioche2006] testis
[josephmedman] epididymis, sem. vescicles, vas deferens.
[niks] oh...the precursor comes from adrenals I guess
[josephmedman] fetal DHT where does it develop?
[bioche2006] god one joeph
[niks] thanks joe..
[josephmedman] well if DHTA sulfate is increased it is of adrendal orgin
[josephmedman] DHT fetal develops in prostate
[bioche2006] rt they r c 19 ketosteroid in urine
[bioche2006] thanks joseph
[josephmedman] what is 5 alpha red. def.?
[bioche2006] no dht hence no dev of extranl male genitlia and prostate
[niks] prostate will be small
[josephmedman] good
[josephmedman] what drug do you give people with BPH?
[niks] finesteride
[bioche2006] i rt
[josephmedman] good
[niks] its 5 alpfa red inhibhitor
[niks] also prazosin
[josephmedman] what is Rx for malignant HTN?
[bioche2006] rt it relaxres ureter
[bioche2006] nifidipen
[josephmedman] nitroprusside
[bioche2006] sorry
[josephmedman] what is MCC of hydronephrosis?
[bioche2006] it relaxes urthera
[bioche2006] stone
[josephmedman] renal stone
[josephmedman] what is MCCOD in cervical cancer?
[niks] ureter obstruction...
[bioche2006] no idea
[niks] oops...
[perhaps] yes that is correct niks
[niks] oh..
[bioche2006] great niks
[niks] haha
[bioche2006] good
[josephmedman] what microbiological organism
[niks] thnx
[josephmedman] is associated
[niks] HPV
[josephmedman] with magnesium ammonium phosphate stones?
[bioche2006] hpv 6 and 11
[perhaps] proteus
[niks] oh..sorry
[bioche2006] proteus
[josephmedman] good perhaps..
[niks] proteus
[perhaps] thanks
[josephmedman] iof you see a mass in the kidney of the child what are you thinking?
[niks] HPv is associated with cervix Ca
[josephmedman] oh ok..
[saimakm] willms
[niks] wilms
[perhaps] wilm
[bioche2006] wilms
[josephmedman] MC mets of renal adenocarcinoma?
[perhaps] lung
[bioche2006] bone
[josephmedman] lung
[josephmedman] MC cancer of renal pelvis?
[josephmedman] bone is for prostate i think
[bioche2006] trans
[josephmedman] correct me if i am wrong..
[bioche2006] rt
[josephmedman] trans is right..
[bioche2006] renal invades blood veseesl
[josephmedman] almost done guys
[niks] yes joe,,...bone for rostate
[bioche2006] so lung look rt
[josephmedman] MCC of oliguria?
[niks] through verterbral plexus..hematogenous spread
[bioche2006] rt niks
[saimakm] trans?
[bioche2006] transtitional
[saimakm] ok
[bioche2006] dehydration mmc of oliguria
[josephmedman] pre renal azotemia
[niks] oh..
[bioche2006] ok
[josephmedman] what type of infection is acute pyelonephritis?
[josephmedman] ascending..
[niks] its due to E coli
[bioche2006] rt
[niks] ascending
[josephmedman] do you see WBC casts in acute cystitits?
[bioche2006] no
[niks] nope
[usmle.doc] no
[josephmedman] what is the MCC of chronic pyelonephritis?
[niks] chronic obstruction due to stone
[usmle.doc] obstrucion
[niks] or reflux?
[josephmedman] vesicourureteral reflux
[bioche2006] reflux
[bioche2006] rt
[niks] oh ok...
[josephmedman] what is the MC organ miliary TB goes to?
[bioche2006] kidney hehehehehehehe
[josephmedman] kidney
[josephmedman] haha
[josephmedman] what is the MC cancer of penis?
[niks] it has to be kidney coz we are discussing renal...hahah
[bioche2006] squamous
[josephmedman] and what is the MC reason?
[bioche2006] phimosis
[saimakm] uncircum
[josephmedman] lack of circumsision
[bioche2006] yeah same
[josephmedman] what is phimosis again bioch?
[josephmedman] oh ok..
[josephmedman] where is cryptorchidism MC location?
[bioche2006] undescedned testis
[bioche2006] ingunal canal
[usmle.doc] inguinal canal
[josephmedman] inguinal canal
[josephmedman] what is MCC of left sided scrotal enlargement?
[bioche2006] high risk of which tumour
[josephmedman] seminoma
[bioche2006] rt
[saimakm] seminoma
[bioche2006] varisoce
[usmle.doc] varicocele
[bioche2006] rt
[saimakm] vari
[josephmedman] spermatic vein comes off what in varicocele?
[josephmedman] left renal vein..
[josephmedman] it is ver common cause of infertility
[bioche2006] goes to left renal
[bioche2006] rt
[josephmedman] MC germ cell tumor?
[perhaps] seminoma?
[saimakm] yolk sac
[bioche2006] seminoam
[josephmedman] here we go niks..
[josephmedman] seminoma
[usmle.doc] seminoma
[josephmedman] where is the most common metastatsis?
[usmle.doc] pre and para aortic lymphnoeds
[bioche2006] rt
[sanya] Hi guys where is the biochem chat going on?
[josephmedman] paraaortic lymph nodes..
[bioche2006] para aotic
[usmle.doc] ok
[josephmedman] what was yoru question b4 niks?
[saimakm] para aortic
[josephmedman] that you thought was the para aortic lymph node?
[niks] hmm...what quest joe...I think I forgot
[josephmedman] oh ok..
[josephmedman] it was way in the beginning
[josephmedman] what is the most common hem. site?
[niks] oh...I just asked...and you ansrwered rightly...
[bioche2006] hem?
[josephmedman] lung
[josephmedman] MC cancer in adult males?
sanya has left the subroom.
[saimakm] lung
[usmle.doc] prostate
[bioche2006] prostate
[josephmedman] it was the horseshoe kidney questions niks
sanya has left the chat.
[josephmedman] what disease is there a loss of inhibin?
[bioche2006] excess fsh
[bioche2006] i dont know woaht does it do
[josephmedman] Klinefelters..
[bioche2006] how
[josephmedman] thats it for today guys
[niks] oh...I'll check that out...I thought its caught below where the abdominal aorta divides...I was no sure though
[saimakm] primary hypogonadism
[josephmedman] i think we covered all the high yield stuff